This effect may be involved in any beneficial role of heart rate lowering agents in preventing coronary heart disease. Slowing the heart rate could potentially decrease the progression of atherosclerosis by reducing the local pro-atherosclerotic vascular environment. If you have an arrhythmia that causes the heart to beat faster than normal, this is known as tachycardia. But many things affect where someone’s heart rate usually lives within that range, including: Fitness: People with good cardiovascular fitness like athletes tend to have lower resting heart. A very high heart rate can indicate a problem with the heart or a blockage in the cardiovascular system, which may lead to a heart attack, stroke, or other serious complications. The average adult’s heart rate ranges from 60 to 100 beats per minute. Heart rate should be considered in every patient with coronary heart disease, especially since it is an easily measurable and reproducible parameter. A normal resting heart rate will be a little different for each person. For instance, the heart rate typically rises during exercise or as a response to stress. Many types of irregular heart rhythms (arrhythmias) can cause tachycardia. The device can if a person were to go into a life-threatening ventricular tachycardia deliver a potentially lifesaving shock to prevent sudden death. When most patients start to exercise, their own heart rate rises and the PVCs or other extra beats disappear at higher heart rates. All these processes induce structural and functional changes of the endothelial cells, which are accumulated over the time in atherosclerosis-prone regions promoting atherosclerosis. Tachycardia (tak-ih-KAHR-dee-uh) is the medical term for a heart rate over 100 beats a minute. Moreover, increased heart rate intensifies the pulsatile motion of the heart and, therefore, the frequency of the periodically changing geometry of the coronary arteries, thereby affecting the local hemodynamic environment. Fit and relaxed horses may occasionally drop a. An elevated heart rate enhances the magnitude and frequency of the tensile stress imposed on the arterial wall and prolongs the exposure of coronary endothelium to the systolic low and oscillatory shear stress. There may be a slight increase in heart rate during inspiration, a normal finding known as sinus arrhythmia. In this review we explore the pathophysiologic mechanisms involved in the pro-atherosclerotic effect of elevated heart rate, apart from its association with sympathetic tone. Several epidemiological studies have reported that an elevated heart rate is associated with coronary atherosclerosis independently of other risk factors.
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